Importantly, such approaches bypass the drug-resistance of cancer cells without provoking the selection of malignant cancer cell lineages. total of 25 M FF exerted corresponding effects on Cx43-mediated gap junctional coupling, EGF production, and ERK1/2 activation in HUVEC/A549 co-cultures. It also directly augmented endothelial barrier function via the interference with focal adhesion kinase (FAK)/RhoA/Rac1-regulated endothelial … Continue reading Importantly, such approaches bypass the drug-resistance of cancer cells without provoking the selection of malignant cancer cell lineages
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